Thus, a combination of these two factors during the acute stages of retinopathy produces marked retinal damage and marked loss of peripheral visual field due to pan retinal photo coagulation. Most patients are referred to their internist for the appropriate medical evaluations. It appears that the more complete the blockage, the more intense the haemorrhages and the enema. Recchia FM, Brown AC. The study authors concluded that high blood pressure and vascular diseases are important risk factors for central retinal vein occlusion, and that blacks have a significantly greater risk for CRVO than other races. Ophthalmology 1981;88:18-25. Late phase of fluoresce in angiography of the same patient as in previous image, showing cystoid pattern of leakage from perifoveal dilated leaking capillary network. While there are similarities in the pathogenesis and clinical nature of these two events, each has unique ethologies, differential diagnosis, management and prognosis. Angiography is usually much less useful than the functional tests during the acute stages of the disease. Other symptoms can include a vague haziness or loss of perception.
Uveitis is inflammation and swelling that occurs in the urea. Weiss GR, Sipperley J, Gaitan BR. Does individual complain of sudden onset of blurred or distorted vision in the upper or lower half or total visual field? act Ophthalmol sand 2002;80:355-67. Graefes Arch Cain Exp Ophthalmol 2002;240:180-96. Zegarra H, gunman FA, Conforto J: The natural course of central retinal vein occlusion.